Homocysteine
Homocysteine is a sulfur-containing amino acid produced during the metabolism of methionine, an essential amino acid. It plays a key role in methylation, detoxification, and cardiovascular function. Elevated homocysteine levels have been associated with increased risk of heart disease, stroke, cognitive decline, and pregnancy complications. Imbalances may be caused by deficiencies in B vitamins (especially B6, B12, and folate), impaired methylation, or genetic variants like MTHFR. Monitoring homocysteine levels can provide valuable insights into cardiovascular, neurological, and metabolic health.
Description
Elevated homocysteine is often linked to an increased risk of cardiovascular disease, impaired detoxification, inflammation, and neurological concerns such as memory issues or mood imbalances.
Common causes include deficiencies in vitamin B6, B12, or folate, genetic polymorphisms (e.g., MTHFR), poor methylation, chronic stress, and kidney dysfunction.
High homocysteine often has no symptoms, but may be associated with fatigue, brain fog, irritability, or numbness and tingling if left unaddressed.
Improving B-vitamin status—especially folate, B6, and B12—through diet or supplementation can help normalize homocysteine. Addressing methylation imbalances may also be necessary.
Because it reflects both cardiovascular and methylation health, tracking homocysteine can help detect silent imbalances that may contribute to chronic disease if left untreated.
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